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Physiology:
GAS
AO1
Selye (1936) universal stress response = adaptive
Non specific - same all stressors
Body attempts to remain balanced through activation / recovery
Stage 1: Alarm reaction, SAM pathway, adrenaline released from adrenal medulla
Stage 2: Resistance, body adapts by resisting stressor, hormones depleted, parasympathetic NS activated to conserve energy. HPA axis releases cortisol
Stage 3: Exhaustion, Adaptation to chronic stressor fails - all resources depleted, symptoms of sympathetic arousal, adrenal glands damaged, stress related illnesses e.g. CHD, depression
AO3
+ Selye research on rats
- Not replicated on monkeys (Mason, 1971)
- Animal extrapolation
Stress & Illness: Cardiovascular
AO1
Cardiovascular disorders = heart & blood vessels e.g. CHD, stroke
Acute stress = high adrenaline - direct effects on heart muscles
Acute stress & football match: Wilbert-Lampen, heart attacks German 1996 World Cup. Day Germany play, cardiac emergencies x2.66
Chronic stress in workplace: Yusuf Interheart Study. 15k people with heart attack & control. Chronic stressors had strong link to heart attack. Stress greater than obesity 3rd after smoking & cholesterol
AO3
- Link appears to be indirect. Orth-Gomer marital stress x3 risk of heart attack but only if pre-CVD
+ Supportive evidence. Song (2019) ppts with stress related disorders compared to siblings/controls. Stress led to 64% greater risk CVD.
- Cause physiological or psychological?
Sources of Stress: Workplace Stress
AO1
Coming soon
AO3
Coming soon
Individual Differences: Personality
AO1
Friedman & Rosenman observed a personality type of ppts with CHD
Type A: Competitive, time urgent, hostile
Type B: Laid back, reflective and tolerant - not linked to CHD
Western Collaborative Group Study: 3000 males, USA, no CHD at start of study. Answered 25 qus to assess personality type. Interviews conducted to incite Type A traits.
8.5 yrs later 257 men had CHD and 70% of men were assessed as Type A. Impatience and hostility -> physiological stress response.
Type C: Pathological niceness - compliant, passive, repress emotions. Linked to cancer.
Dattore (1980) studied 200 veterans of Vietnamese War - 75 cancer patients vs other diagnoses. Cancer ppts reported significantly greater suppression and fewer depression symptoms.
AO3
+ Real world application. Type A change behaviours after CHD and live longer
- Too broad? Linked to hostility mostly (Carmelli 1991)
- Type C and cancer link not reliable finding - mediating factors
- Too binary of A/B. Personality is more nuanced.
Managing Stress: Stress Inoculation Therapy
AO1
Coming soon
AO3
Coming soon
Managing Stress: Social Support
AO1
Coming soon
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Coming soon
Physiology: Stress Response
AO1
Acute (short term): SAM pathway -> Hypothalamus -> Activates sympathetic branch NS -> Triggers adrenal medulla release adrenaline / noradrenaline -> prepares body F or F. When passed parasympathetic NS activated.
Chronic (long term): HPA system activated. Hypothalamus -> corticotropin releasing factor (CRF) -> pituitary gland releases ACTH -> adrenal cortex releases cortisol -> glucose metabolism, restores energy, suppresses immune system. HPA self regulating. High levels of cortisol reduces CRF & ACTH reducing cortisol.
AO3
+ Real life: Addison's disease - cortisol replacement therapy
- Ignores psychological factors. Speisman (1964) medical procedure
- Gender bias acute stress - Taylor's "tend and befriend"
Sources of Stress: Life Changes
AO1
Life changes are important but infrequent life events
Need psychological adjustments
Bigger change = more stress
They are cumulative & can apply to + and - events
Holmes & Rahe Social Readjustment Rating Scale gives number of life changing units - higher the value = more stress e.g. divorce is 73. Self report (12 mths)
More LCU, more stress.
Rahe (1972) claimed that <150 LCU = reasonable health, 150-3000 LCUs 50% chance of illness, >300 LCUs 80% chance illness in next yr
Rahe (1970) US Navy Study +ve LCU in last 6 mths & illness on ship
AO3
+ Evidence: Lietzen (2011) 16k asthma free ppts. LCU predictor.
- Ignores individual differences - subjective opinion of LCU needed
- +ve and -ve events not the same
- Ignores third variables
Measuring Stress:
Self Report
AO1
Coming soon
AO3
Coming soon
Individual Differences: Hardiness
AO1
Coming soon
AO3
Coming soon
Managing Stress: Biofeedback
AO1
Coming soon
AO3
Coming soon
Stress & Illness: Immunosuppresion
AO1
Directly: cortisol produced by the HPA inhibits production of immune cells
Indirectly: stress can lead to unhelpful lifestyle choices e.g. drinking that -ve effects immune functioning
Chronic stress of exams: Kiecolt-Glaser medical students, decrease in natural killer and killer T cells when highly stressed. Greatest when student lonely or other stress
Chronic stress of caring for ill relative: Kiecolt-Glaser compared carers (of Alzheimers) with control. Over 13 months carers had weaker cell based immune response & more infectious diseases/ depression (32%) compared to control group (6%).
AO3
- Acute stress can have immunoenhancing effects
+ Real life application: stress hormones before life stressor
- Methodological issues in lab
Sources of Stress: Daily Hassles
AO1
Daily hassles include minor inconveniences e.g. lost keys and greater pressures e.g. no time
Primary appraisal - assess subjectively how psychologically threatening something is
Secondary appraisal - if threatening we assess how equipped we are to deal with it
Hassles and Uplifts Scale (HSUP) = self report. How many / severity of halles & how many uplifts
Life changes = indirect effects = distal sources of stress
Daily hassles = direct and immediate threat = proximal sources of threat
Kanner et al (1981) 100 ppts did HSUP every mth x 9
Life changes scale one month before study and at end
Hopkins symptom checklist for anxiety & depression
+ve correlation hassles frequency & symptoms
Hassles greater predictor than life changes.
Measuring Stress: Physiological
AO1
Coming soon
AO3
Coming soon
Managing Stress: Drug Therapies
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Coming soon
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Managing Stress: Gender Differences
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Coming soon
AO3
Coming soon
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